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[[short term rewards|short term rewards]] & [[long term rewards]].
==Dopamine==
[[Dopamine]] is involved in reward. Dopamine appear to play a central role in cost-benefit analysis<ref>Phillips PEM, Walton ME, Jhou TC. Calculating utility: Preclinical evidence for cost– benefit analysis by mesolimbic dopamine. Psychopharmacology. 2007;191: 483– 495.</ref>. It seems to be part of the [[rewards|reward sytem]] There appear to be multiple dopamine-sensitive decision regions.
[[Dopamine|Dopamine]] depletion in [[Ventral stratium|ventral striatum]] reduces propensity for physical effort <ref>Salamone JD, Correa M, Farrar A, Mingote SM. Effort-related functions of nucleus accumbens dopamine and associated forebrain circuits. Psychopharmacology. 2007;191: 461– 482.</ref> D1 ([[dopamine]] 1) receptor blockade in [[ACC]] reduces preference for expending effort for rats<ref>Schweimer J, Hauber W. Dopamine D1 receptors in the anterior cingulate cortex regulate effort-based decision-making. Learning & Memory. 2006;13: 777–782</ref>.It is non-discriminative between reward types, dopaminergic firing in [[VTA]] does appear to reflect subjective (action) value with integrated responses to both delay and reward amount<ref>Roesch MR, Calu DJ, Schoenbaum G. Dopamine neurons encode the better option in rats deciding between differently delayed or sized rewards. Nature Neuroscience. 2007;10: 1615– 1624.</ref>. Dopaminergic neurons send diffuse projections to [[striatum]] (nigrostriatal pathway) and prefrontal cortex (mesocortical pathway) and thereby transmit a pleasure values or [[learning|teaching]] signal to a variety of brain regions, for learning, stimulus evaluation, and directed action. <ref>Volkow ND, Wang GJ, Telang F, et al. Dopamine increases in striatum do not elicit craving in cocaine abusers unless they are coupled with cocaine cues. NeuroImage. 2008;39: 1266– 1273)</ref>.
==Hormones effects==
There are substantial hormonal influences on behavior. For example, circulating hormones such as leptin and ghrelin act as satiety and hunger signals, reporting the status of body energy reserves (e.g. adipose tissue), energy requirements, and acute nutrient intake to hypothalamic and midbrain targets in the central nervous system that regulate feeding behavior<ref>Korotkova, Sergeeva, Eriksson et al., 2003</ref>. They also act on brain regions (in particular dopaminoceptive areas) implicated in human decision-making<ref>Hommel, Trinko, Sears et al., 2006</ref><ref>Krügel, Schraft, Kittner et al., 2003</ref>.
==References==